Matrix metalloproteinases: not-so-innocent bystanders in heart failure
- PMID: 11018069
- PMCID: PMC381429
- DOI: 10.1172/JCI11263
Matrix metalloproteinases: not-so-innocent bystanders in heart failure
Comment on
-
Disruption of the myocardial extracellular matrix leads to cardiac dysfunction.J Clin Invest. 2000 Oct;106(7):857-66. doi: 10.1172/JCI8040. J Clin Invest. 2000. PMID: 11018073 Free PMC article.
References
-
- Hunter JJ, Chien KR. Signaling pathways for cardiac hypertrophy and failure. N Engl J Med. 1999;341:1276–1283. - PubMed
-
- Spinale FG, Coker ML, Bond BR, Zellner JL. Myocardial matrix degradation and metalloproteinase activation in the failing heart: a potential therapeutic target. Cardiovasc Res. 2000;46:225–238. - PubMed
-
- Ross RS, et al. Beta1 integrins participate in the hypertrophic response of rat ventricular myocytes. Circ Res. 1998;82:1160–1172. - PubMed
-
- Coral-Vazquez R, et al. Disruption of the sarcoglycan-sarcospan complex in vascular smooth muscle: a novel mechanism for cardiomyopathy and muscular dystrophy. Cell. 1999;98:465–474. - PubMed
-
- Spinale FG, et al. Matrix metalloproteinase inhibition during the development of congestive heart failure: effects on left ventricular dimensions and function. Circ Res. 1999;85:364–376. - PubMed