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Comment
. 2001 Jun;48(6):871-2.
doi: 10.1136/gut.48.6.871.

Helicobacter pyroli infection and acid secretion in patients with duodenal ulcer in Japan

T Chiba, T Watanabe, T Ito
Comment

Helicobacter pyroli infection and acid secretion in patients with duodenal ulcer in Japan

T Chiba et al. Gut. 2001 Jun.
No abstract available

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Figures

Figure 1
Figure 1
Effects of Helicobacter pylori eradication on maximum acid output (MAO) in Japanese patients with duodenal (DU) and gastric (GU) ulcers. MAO in response to pentagastrin (6 µg) was measured before and six months after H pylori eradication. *p<0.05 v before eradication.
Figure 2
Figure 2
Correlation between decrease in serum gastrin concentration (−Δgastrin) and increase in maximum acid output (ΔMAO) in response to pentagastrin (6 µg) by Helicobacter pylori eradication in Japanese patients with duodenal (DU) and gastric (GU) ulcers. Groups A, B, and C show DU patients in whom MAO decreased (more than 1 mEq/h), did not change, and increased (more than 1 mEq/h) after eradication, respectively.
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