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. 2001 Feb;7(1):136-9.
doi: 10.3748/wjg.v7.i1.136.

Methylation status of c-fms oncogene in HCC and its relationship with clinical pathology

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Methylation status of c-fms oncogene in HCC and its relationship with clinical pathology

J Cui et al. World J Gastroenterol. 2001 Feb.
No abstract available

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Figures

Figure 1
Figure 1
Southern blot of HCC tissue genome DNA digested with Hpa II/Msp I. Lane 1, 3, 5, 7, 9, 11 Msp I digestion; Lane 2, 4, 6, 8, 10, 12 Hpa II digestion; Lane 13 λ DNA/Hind III markers; Lane 1, 2 normal control hepatic tissue; Lane 3-12 HCC tissue; Lane 8, 10 hybridizing bands of less than 6.5 kb in size appeared after Hpa II digestion
Figure 2
Figure 2
Southern blot of circumª²cancer liver tissue genome DNA digested with Hpa II/Msp I. Lane 1, 3, 5, 7, 9, 11 Msp I digestion; Lane 2, 4, 6, 8, 10, 12 Hpa II digestion; Lane 13 λ DNA/Hind III markers; Lane 1, 2 normal control hepatic tissue; Lane 3-12 circum-cancer liver tissue; Lane 4 hybridizing bands of less than 6.5 kb in size appeared after Hpa II digestion

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