Knockin' on the door of general anesthetic mechanisms: but will U.S. researchers be shut out?

Affiliations

Editorial

Neil L Harrison. Anesth Analg. 2003 Sep.

. 2003 Sep;97(3):616-618.

doi: 10.1213/01.ANE.0000086120.30172.12.

¹ C.V. Starr Laboratory of Molecular Neuropharmacology, Department of Anesthesiology, Weill Medical College of Cornell University, New York, New York.

No abstract available

Comment on

Inhaled anesthetics and immobility: mechanisms, mysteries, and minimum alveolar anesthetic concentration.
Sonner JM, Antognini JF, Dutton RC, Flood P, Gray AT, Harris RA, Homanics GE, Kendig J, Orser B, Raines DE, Trudell J, Vissel B, Eger EI 2nd. Sonner JM, et al. Anesth Analg. 2003 Sep;97(3):718-740. doi: 10.1213/01.ANE.0000081063.76651.33. Anesth Analg. 2003. PMID: 12933393 Review.

1. Sonner JM, Antognini JF, Dutton RC, et al. Inhaled anesthetics and immobility: mechanisms, mysteries, and MAC. Anesth Analg 2003; 97: 718–40.
1. Harrison NL. Ion channels take center stage: twin spotlights on two anesthetic targets. Anesthesiology 2000; 92: 936–8.
1. Franks N, Lieb W. Molecular mechanisms of general anaesthesia. Nature 1982; 300: 487–93.
1. Franks NP, Lieb WR. Do general anaesthetics act by competitive binding to specific receptors? Nature 1984; 310: 599–601.
1. Franks NP, Lieb WR. Molecular and cellular mechanisms of general anaesthesia. Nature 1994; 367: 607–14.