doi: 10.1093/brain/75.3.408.
A peculiar form of peripheral neuropathy; familiar atypical generalized amyloidosis with special involvement of the peripheral nerves
- PMID: 12978172
- DOI: 10.1093/brain/75.3.408
Item in Clipboard
A peculiar form of peripheral neuropathy; familiar atypical generalized amyloidosis with special involvement of the peripheral nerves
Brain.
1952 Sep.
No abstract available
Comment in
-
Modified protocol for the application of botulinum toxin type A for hyperhidrosis of the nose.Int J Dermatol. 2013 Nov;52(11):1400-1. doi: 10.1111/j.1365-4632.2011.05150.x. Int J Dermatol. 2013. PMID: 24164155 No abstract available.
Similar articles
-
[Ultrastructural study of amyloid neuropathy with special reference to changes of the peripheral nerves in asymptomatic siblings].Rinsho Shinkeigaku. 1974 Mar;14(3):266-74. Rinsho Shinkeigaku. 1974. PMID: 4371501 Japanese. No abstract available.
-
[HEART CHANGES IN THE FAMILIAL TYPE OF PARAMYLOIDOSIS WITH PERIPHERAL NEUROPATHY].Z Kreislaufforsch. 1963 Nov;52:1066-78. Z Kreislaufforsch. 1963. PMID: 14097990 German. No abstract available.
-
Peripheral nerve involvement in primary systemic AL amyloidosis: a clinical and electrophysiological study.Eur J Neurol. 2011 Apr;18(4):604-10. doi: 10.1111/j.1468-1331.2010.03215.x. Epub 2010 Sep 23. Eur J Neurol. 2011. PMID: 20860753
-
Amyloid-related neuropathies.Zentralbl Allg Pathol. 1990;136(6):517-23. Zentralbl Allg Pathol. 1990. PMID: 2177943 Review.
-
[Lesions of peripheral and cranial nerves in amyloidosis].Klin Med (Mosk). 1984 Apr;62(4):14-27. Klin Med (Mosk). 1984. PMID: 6330435 Review. Russian. No abstract available.
Cited by
-
Silencing human genetic diseases with oligonucleotide-based therapies.Hum Genet. 2013 May;132(5):481-93. doi: 10.1007/s00439-013-1288-1. Epub 2013 Mar 14. Hum Genet. 2013. PMID: 23494242 Review.
-
Primary neuropathic amyloidosis in three brothers.Br Med J. 1974 Jan 12;1(5897):65-6. doi: 10.1136/bmj.1.5897.65. Br Med J. 1974. PMID: 4812395 Free PMC article.
-
Haplotype analysis of familial amyloidotic polyneuropathy. Evidence for multiple origins of the Val----Met mutation most common to the disease.Hum Genet. 1989 Apr;82(1):9-13. doi: 10.1007/BF00288262. Hum Genet. 1989. PMID: 2714785
-
Deposition of transthyretin in early stages of familial amyloidotic polyneuropathy: evidence for toxicity of nonfibrillar aggregates.Am J Pathol. 2001 Dec;159(6):1993-2000. doi: 10.1016/s0002-9440(10)63050-7. Am J Pathol. 2001. PMID: 11733349 Free PMC article.
-
Potentially amyloidogenic conformational intermediates populate the unfolding landscape of transthyretin: insights from molecular dynamics simulations.Protein Sci. 2010 Feb;19(2):202-19. doi: 10.1002/pro.289. Protein Sci. 2010. PMID: 19937650 Free PMC article.
MeSH terms
LinkOut - more resources
Full Text Sources
Other Literature Sources
Molecular Biology Databases
