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. 1962 Jun:1:187-203.
doi: 10.1016/0014-4800(62)90020-5.

Histopathologic alterations associated with the transplanted homologous dog liver

Histopathologic alterations associated with the transplanted homologous dog liver

D R BROCK et al. Exp Mol Pathol. 1962 Jun.
No abstract available

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Figures

Fig. 1
Fig. 1
Liver from dog surviving 10 hours. Note portal venous and lymphatic engorgement characteristic of outflow block, × 43.
Fig. 2
Fig. 2
Liver from dog surviving 10 hours illustrating Kupfer cell activation, × 263.
Fig. 3
Fig. 3
Arteriole of liver showing endothelial and perithelial proliferation with focal hyaline degeneration, × 263.
Fig. 4
Fig. 4
Liver: 10 day survival, × 263. Periportal and parenchymal infiltrate is primarily plasmacytic.
Fig. 5
Fig. 5
General hepatic architecture: (a) 9 day survival, × 42; (b) another dog, 8 day survival, × 42.
Fig. 5
Fig. 5
General hepatic architecture: (a) 9 day survival, × 42; (b) another dog, 8 day survival, × 42.
Fig. 6
Fig. 6
Lymph node from dog surviving 20 days: (a) cortical depletion, × 82; (b) cortical cell type primarily mononuclear and plasmacytic, × 232.
Fig. 6
Fig. 6
Lymph node from dog surviving 20 days: (a) cortical depletion, × 82; (b) cortical cell type primarily mononuclear and plasmacytic, × 232.
Fig. 7
Fig. 7
Kidney: (a) plasmacytic aggregation, × 97; (b) tubular basement membrane activation, × 97.
Fig. 8
Fig. 8
Perirenal plasmacytic and mononuclear infiltration, × 97.
Fig. 9
Fig. 9
Lung. Focal alveolar wall proliferation, mononuclear cell infiltrate and giant cell formation, × 225.
Fig. 10
Fig. 10
Bone marrow. Note increased plasmacytes, × 392.
Fig. 11
Fig. 11
Small intestine. Mucosal slough and mononuclear infiltration, × 42.

References

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