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Review
. 2004 Sep;6(5):379-84.
doi: 10.1007/s11886-004-0041-8.

Drugs that cause Torsades de pointes and increase the risk of sudden cardiac death

Affiliations
Review

Drugs that cause Torsades de pointes and increase the risk of sudden cardiac death

Deborah L Wolbrette. Curr Cardiol Rep. 2004 Sep.

Abstract

Torsades de pointes (TdP) is a potentially life-threatening arrhythmia associated with not only antiarrhythmic drugs, but noncardiac drugs of many different classes. All these drugs prolong the QT interval by their blocking of the potassium channel I(Kr), and many are metabolized by the cytochrome P450 isoenzyme CYP3A4. Polypharmacy with other drugs utilizing the same enzyme, or inhibiting CYP3A4, can lead to TdP. A consistent finding of all the QT-prolonging drugs is predominance of TdP in women. Other risk factors for QT prolongation and TdP include hypokalemia, congestive heart failure, and structural heart disease. Knowledge of potential drug interactions and other risk factors for TdP can help in reducing the number of adverse events associated with the use of QT-prolonging drugs.

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References

    1. Drugs. 2002;62(11):1649-71 - PubMed
    1. Pacing Clin Electrophysiol. 2001 Oct;24(10):1575-6 - PubMed
    1. JAMA. 1993 Mar 24-31;269(12):1532-6 - PubMed
    1. Am J Psychiatry. 2001 Nov;158(11):1774-82 - PubMed
    1. J Cardiovasc Electrophysiol. 1998 Oct;9(10):1109-13 - PubMed

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