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Case Reports
. 2004 Nov-Dec;25(10):1685-7.

Early uncoupling of cerebral blood flow and metabolism after bilateral thalamic infarction

Beau M Ances  1 , David S Liebeskind, Andrew B Newberg, Dina A Jacobs, Abass Alavi
Affiliations
Case Reports

Early uncoupling of cerebral blood flow and metabolism after bilateral thalamic infarction

Beau M Ances et al. AJNR Am J Neuroradiol. 2004 Nov-Dec.

Abstract

Cerebral blood flow and metabolism may be uncoupled in the early phases after stroke onset. Prior reports of bilateral thalamic stroke have described subsequent coupling of blood flow and metabolism during the chronic stage. We chronicled the evolving relationship of blood flow and metabolism with concomitant single photon emission CT and positron emission tomography from the subacute to chronic phase following bilateral thalamic infarction.

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Figures

F<sc>ig</sc> 1.
Fig 1.
Noncontrast CT on admission reveals a chronic left frontal infarct without acute ischemic changes.
F<sc>ig</sc> 2.
Fig 2.
Emergent angiogram of the basilar artery (anteroposterior view) demonstrates absence of arterial occlusion.
F<sc>ig</sc> 3.
Fig 3.
Subacute bilateral paramedian thalamic infarction depicted on noncontrast CT.
F<sc>ig</sc> 4.
Fig 4.
Concomitant SPECT (A) and FDG-PET (B) during the subacute phase illustrates thalamic luxury perfusion and cortical diaschisis.
F<sc>ig</sc> 5.
Fig 5.
At 6 months from stroke onset, concomitant SPECT (A) and FDG-PET (B) detail normal thalamic coupling and resolution of diffuse cortical diaschisis.
See this image and copyright information in PMC

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