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Editorial
. 2005 Apr;91(4):421-2.
doi: 10.1136/hrt.2004.039065.

Coronary recanalisation, myocardial viability, and ventricular remodelling after infarction

Editorial

Coronary recanalisation, myocardial viability, and ventricular remodelling after infarction

P G Camici. Heart. 2005 Apr.

Abstract

It is important to identify the mechanisms that determine the progression to left ventricular remodelling after an acute myocardial infarction, in order that patients can be treated before the development of overt heart failure.

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Figures

Figure 1
Figure 1
Myocardial β adrenoceptor downregulation soon after acute myocardial infarction predicts left ventricular (LV) dilatation. β-AR, myocardial β adrenoceptor density; LV improved, those patients (n  =  41) whose left ventricular volumes were found to be decreased six months after infarction; LV dilated, those patients (n  =  20) whose left ventricular volumes were found to be increased six months after infarction. Adapted from Spyrou and colleagues.

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References

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