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Comment
. 2005 Oct 25;173(9):1049-50.
doi: 10.1503/cmaj.051212.

How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?

Thomas J Louie  1
Affiliations
Comment

How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?

Thomas J Louie. CMAJ. .
No abstract available

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References

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    1. Spigalia P, Mastrantonio P. Molecular analysis of the pathogenicity locus and polymorphism in the putative negative regulator of toxin production (TcdC) among Clostridium difficile clinical isolates. J Clin Microbiol 2002;40:3470-75. - PMC - PubMed
    1. Pépin J, Valiquette L, Alary ME, Villemure P, Pelletier A, Forget K, et al. Clostridium difficile–associated diarrhea in a region of Quebec from 1991 t6 2003: a changing pattern of disease severity. CMAJ 2004;171(5):466-72. - PMC - PubMed
    1. Pépin J, Valiquette L, Cossette B. Mortality attributable to nocosomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec. CMAJ 2005;173(9):1037-41. - PMC - PubMed
    1. McDonald LC, Killgore GE, Thompson A, et al and the C. difficile investigation team. Emergence of an epidemic strain of Clostridium difficile in the United States 2001-4: potential role for virulence factors and antimicrobial resistance traits. Abstract LB-2, 42nd Annual Meeting of the Infectious Disease Society of America, Boston, October 2004.

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