Novel mechanism for impaired nitrergic relaxation in achalasia
- PMID: 16474101
- PMCID: PMC1856096
- DOI: 10.1136/gut.2005.078402
Novel mechanism for impaired nitrergic relaxation in achalasia
Abstract
New insights into the pathogenesis of achalasia indicate that incubation with serum from patients with achalasia leads to altered neurochemical coding of the myenteric plexus and impairs the nitrergic response to nerve stimulation
Conflict of interest statement
Conflict of interest: None declared.
Comment on
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Serum from achalasia patients alters neurochemical coding in the myenteric plexus and nitric oxide mediated motor response in normal human fundus.Gut. 2006 Mar;55(3):319-26. doi: 10.1136/gut.2005.070011. Epub 2005 Aug 16. Gut. 2006. PMID: 16105888 Free PMC article.
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