Autoimmunity in myocardial infarction
- PMID: 16837084
- DOI: 10.1016/j.ijcard.2006.05.009
Autoimmunity in myocardial infarction
Abstract
Myocardial infarction is associated with an immune response. Physiological inflammation response causes self-repair and protection, while pathological autoimmune response leads to ventricular remodeling and heart failure. Laying emphasis on regulating the immune function may become a new target for the prevention of heart failure after myocardial infarction. This review focuses on the mechanism of immune-mediated ventricular remodeling and the immune therapy after myocardial infarction.
Similar articles
-
[Prognostic significance of typical immune reactions in myocardial infarction].Ter Arkh. 2008;80(1):32-7. Ter Arkh. 2008. PMID: 18326224 Russian.
-
Targeted deletion of nuclear factor kappaB p50 enhances cardiac remodeling and dysfunction following myocardial infarction.Circ Res. 2009 Mar 13;104(5):699-706. doi: 10.1161/CIRCRESAHA.108.189746. Epub 2009 Jan 24. Circ Res. 2009. PMID: 19168865
-
Post-infarct remodelling: contribution of wound healing and inflammation.Cardiovasc Res. 2009 Feb 15;81(3):474-81. doi: 10.1093/cvr/cvn292. Epub 2008 Oct 31. Cardiovasc Res. 2009. PMID: 18977766 Free PMC article. Review.
-
Toll-like receptor 4 mediates maladaptive left ventricular remodeling and impairs cardiac function after myocardial infarction.Circ Res. 2008 Feb 1;102(2):257-64. doi: 10.1161/CIRCRESAHA.107.158220. Epub 2007 Nov 15. Circ Res. 2008. PMID: 18007026
-
Inflammation following acute myocardial infarction: Multiple players, dynamic roles, and novel therapeutic opportunities.Pharmacol Ther. 2018 Jun;186:73-87. doi: 10.1016/j.pharmthera.2018.01.001. Epub 2018 Jan 9. Pharmacol Ther. 2018. PMID: 29330085 Free PMC article. Review.
Cited by
-
The Influence of Metabolic Risk Factors on the Inflammatory Response Triggered by Myocardial Infarction: Bridging Pathophysiology to Treatment.Cells. 2024 Jun 29;13(13):1125. doi: 10.3390/cells13131125. Cells. 2024. PMID: 38994977 Free PMC article. Review.
-
Emergence of dendritic cells in the myocardium after acute myocardial infarction - implications for inflammatory myocardial damage.Int J Biomed Sci. 2010 Mar;6(1):27-36. Int J Biomed Sci. 2010. PMID: 23675173 Free PMC article.
-
T-cells in myocardial infarction: Culprit instigators or mere effectors?World J Cardiol. 2018 Oct 26;10(10):123-126. doi: 10.4330/wjc.v10.i10.123. World J Cardiol. 2018. PMID: 30386489 Free PMC article.
-
Myeloid interleukin-4 receptor α is essential in postmyocardial infarction healing by regulating inflammation and fibrotic remodeling.Am J Physiol Heart Circ Physiol. 2021 Jan 1;320(1):H323-H337. doi: 10.1152/ajpheart.00251.2020. Epub 2020 Nov 8. Am J Physiol Heart Circ Physiol. 2021. PMID: 33164548 Free PMC article.
-
Cells of the Immune System in Cardiac Remodeling: Main Players in Resolution of Inflammation and Repair After Myocardial Infarction.Front Immunol. 2021 Apr 2;12:664457. doi: 10.3389/fimmu.2021.664457. eCollection 2021. Front Immunol. 2021. PMID: 33868315 Free PMC article. Review.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Medical