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Editorial
. 2007 May 1;92(9):3332-4.
doi: 10.1529/biophysj.106.100362. Epub 2007 Feb 2.

Response to bistability in apoptosis: roles of bax, bcl-2, and mitochondrial permeability transition pores

Thomas Eissing, Steffen Waldherr, Frank Allgöwer, Peter Scheurich, Eric Bullinger
Editorial

Response to bistability in apoptosis: roles of bax, bcl-2, and mitochondrial permeability transition pores

Thomas Eissing et al. Biophys J. .
No abstract available

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References

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    1. Stelling, J., U. Sauer, Z. Szallasi, F. J. Doyle 3rd, and J. Doyle. 2004. Robustness of cellular functions. Cell. 118:675–685. - PubMed
    1. Danial, N. N., and S. J. Korsmeyer. 2004. Cell death: critical control points. Cell. 116:205–219. - PubMed
    1. Eissing, T., H. Conzelmann, E. D. Gilles, F. Allgöwer, E. Bullinger, and P. Scheurich. 2004. Bistability analyses of a caspase activation model for receptor-induced apoptosis. J. Biol. Chem. 279:36892–36897. - PubMed
    1. Legewie, S., N. Blüthgen, and H. Herzel. 2006. Mathematical modeling identifies inhibitors of apoptosis as mediators of positive feedback and bistability. PLoS Comput. Biol. 2:e120. - PMC - PubMed

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