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. 2004 Mar 30;10 Suppl 1(Suppl 1):97-100.
doi: 10.1177/15910199040100S115. Epub 2008 Jun 9.

Stenting for atherosclerotic stenosis of the intracranial or skull base cerebral arteries. Effectiveness and problems

T Harakuni  1 , A Hyodo, T Shingaki, M Kugai, T Kinjyo, T Kinjyo, H Tsuchida, K Sugimoto, Y Yoshii, Y Matsumaru
Affiliations

Stenting for atherosclerotic stenosis of the intracranial or skull base cerebral arteries. Effectiveness and problems

T Harakuni et al. Interv Neuroradiol. .

Abstract

Since May 1992, we have performed percutaneous transluminal angioplasty (PTA) or stenting 70 times for 65 lesions in 62 patients with atherosclerotic stenosis of the intracranial or skull base cerebral arteries. Stenting was carried out nine times for nine lesions in nine cases. Stenting was performed on patients with an average age of 62. The patients were eight men and one woman. The stenotic lesions involved the internal carotid artery (petrous portion) in four cases, the internal carotid artery (cavernous portion) in two cases, the internal carotid artery (supraclinoid portion) in one case, the middle cerebral artery (M1) in one case, and the vertebral artery (V4) in one case. The degree of stenosis ranged from 70% to 99%, with a mean of 80%. A stent for coronary arteries was used in all cases. After PTA was carried out in four cases, their initial extension was inadequate, and dissection was performed in five cases after PTA. As for the results of the treatment, subarachnoid haemorrhage occurred in one case due to perforation by the guidewire, and a major deficit was accepted. During the operation, asymptomatic cerebral infarction by distal embolism occurred in one case. Although obstruction of the lesion occurred three months after treatment in one case, symptoms did not appear. Stents used for atherosclerotic stenosis of the intracranial or skull base cerebral arteries still do not have sufficient performance. Although the stenting had problems, such as a prolonged patent, in the present condition, it was effective in terms of recovery from complications due to PTA.

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Figures

Figure 1
Figure 1
R.CAG, A) Stenosis was recognized in the intracranial internal carotid artery (supraclinoid portion). B) The intracranial internal carotid artery after PTA was imaged. The stenosis has not improved.
Figure 2
Figure 2
A) The stent (S670; 3.5 mm x 10 mm) was put on the stenosis. B) The stenosis was dilated after stenting.
See this image and copyright information in PMC

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