Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
Review
. 2010 Dec;35(12):1875-9.
doi: 10.1007/s11064-010-0298-9. Epub 2010 Oct 27.

Neurochemistry of stress. An overview

Affiliations
Review

Neurochemistry of stress. An overview

Nicole Baumann et al. Neurochem Res. 2010 Dec.

Abstract

Stress is a word that is used very commonly. It is generally employed to design unpleasant phenomena, although it is related to a function necessary to our life. Stress in itself is not a disease. Stress is not an aggression. It is an adaptative response of our body to any demand. Nothing can be done without stress. Stress gives rise to a mobilization of our body to succeed in a group of activities necessary to individual and social life. It favors our dynamism and creativity. But this aptitude can attain its limits, when the solicitations we receive are above what we are able to perform, both in relation to our mental and physical capabilities. The brain controls the systems involved in stress. The main areas are the prefrontal cortex, the limbic system (which comprises the hippocampus and the amygdala) and the hypothalamus. Relations between the prefrontal cortex and the limbic system are important for the planification of action. The main systems of regulation are the sympathetic and parasympathetic systems, the neuro-endocrine system and last but not least the immune system. There is a relation between all our organs and the brain. The genetic aspects and the influences of our past experiences, both during childhood and in adult life, are envisaged.

PubMed Disclaimer

References

    1. Trends Neurosci. 2001 Nov;24(11):671-3 - PubMed
    1. J Clin Endocrinol Metab. 1946 Feb;6:117-230 - PubMed
    1. J Immunol. 2007 Sep 15;179(6):4249-54 - PubMed
    1. Science. 2003 Jul 18;301(5631):386-9 - PubMed
    1. Nat Rev Neurosci. 2009 Jun;10(6):434-45 - PubMed

Publication types

MeSH terms

LinkOut - more resources