Airway hyperresponsiveness in asthma
- PMID: 2104859
- DOI: 10.1080/21548331.1990.11703899
Airway hyperresponsiveness in asthma
Abstract
Some asthma triggers exert their major action through cellular inflammatory mechanisms, with resultant airway hyperreactivity to both allergenic and nonallergenic stimuli. Other triggers are simply bronchospastic, so that they do not increase vulnerability. Anti-inflammatory treatments are more important than purely bronchodilator therapy is for long-term control of asthma.
Similar articles
-
Epithelial dysfunction and airway hyperreactivity in asthma.Prog Clin Biol Res. 1988;263:317-29. Prog Clin Biol Res. 1988. PMID: 2898153 Review.
-
Plates and anti-asthma drugs.Pneumonol Pol. 1988 Oct;56(10):457-62. Pneumonol Pol. 1988. PMID: 3254508 No abstract available.
-
Airway hyperresponsiveness. Mechanisms in experimental models.Chest. 1989 Nov;96(5):1165-8. doi: 10.1378/chest.96.5.1165. Chest. 1989. PMID: 2553345 Review. No abstract available.
-
Roles of hyperresponsiveness and airway inflammation in bronchial asthma.Respiration. 1989;55(2):65-74. doi: 10.1159/000195708. Respiration. 1989. PMID: 2672206 Review.
-
[New aspects in the pathogenesis of bronchial asthma. Bronchial hyperreactivity].Med Klin (Munich). 1988 Feb 26;83(4):142-8. Med Klin (Munich). 1988. PMID: 3130556 Review. German. No abstract available.
Cited by
-
Activities of superoxide dismutases and NADPH oxidase in neutrophils obtained from asthmatic and normal donors.Inflammation. 1993 Jun;17(3):361-70. doi: 10.1007/BF00918997. Inflammation. 1993. PMID: 8392494
-
Neutrophils from asthmatics exhibit diminished responsiveness to 2-chloroadenosine which is reversed by theophylline. Evidence for a cyclic-AMP-independent pathway on human neutrophils.Inflammation. 1992 Apr;16(2):101-16. doi: 10.1007/BF00918951. Inflammation. 1992. PMID: 1317357