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. 2011 Jul;5(7):e1146.
doi: 10.1371/journal.pntd.0001146. Epub 2011 Jul 26.

Cystic echinococcosis: chronic, complex, and still neglected

Collaborators, Affiliations

Cystic echinococcosis: chronic, complex, and still neglected

Enrico Brunetti et al. PLoS Negl Trop Dis. 2011 Jul.
No abstract available

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Conflict of interest statement

The authors have declared that no competing interests exist.

Figures

Figure 1
Figure 1. Life cycle of Echinococcus granulosus in a community of the Middle Atlas region, Morocco.
(We thank M. Kachani, College of Veterinary Medicine, Western University of Health Sciences, for the pictures.)
Figure 2
Figure 2. Severe and life threatening complications of CE.
(A) Biliary obstruction/obstructive cholangitis due to biliary fistulas. (B) Liver abscess formation due to secondary bacterial infection of cysts. (C) Cyst rupture (*) followed by anaphylaxis and secondary echinococcosis. (D) Cysts exerting pressure on vital neighbouring structures (e.g., liver veins resulting in Budd-Chiari Syndrome). (E) Embolism of the right pulmonary artery (arrow) caused by cardiac CE and vascular invasion. (F) CE infestation of the posterior wall of the left heart replacing the myocardial layer at the base of the heart. (We thank W. Hosch, Department of Radiology, and A. Stiehl, Department of Gastroenterology, University Hospital Heidelberg, for the images.).
Figure 3
Figure 3. Comparison of Gharbi's and WHO-IWGE ultrasound classifications of CE cysts.
CL, as a potentially parasitic cyst, was not in Gharbi's classification and needs to be differentiated from non-parasitic cysts. This may also happen with CE1 cysts, when the double layer sign is not evident. Also, WHO CE3b had not been explicitly described by Gharbi but could likely be classified as Type III.

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