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. 2011 Oct 21:5:116.
doi: 10.3389/fnins.2011.00116. eCollection 2011.

Energy drinks and the neurophysiological impact of caffeine

Leeana Aarthi Bagwath Persad  1
Affiliations

Energy drinks and the neurophysiological impact of caffeine

Leeana Aarthi Bagwath Persad. Front Neurosci. .

Abstract

Caffeine is the most widely used psychoactive stimulant with prevalent use across all age groups. It is a naturally occurring substance found in the coffee bean, tea leaf, the kola nut, cocoa bean. Recently there has been an increase in energy drink consumption leading to caffeine abuse, with aggressive marketing and poor awareness on the consequences of high caffeine use. With caffeine consumption being so common, it is vital to know the impact caffeine has on the body, as its effects can influence cardio-respiratory, endocrine, and perhaps most importantly neurological systems. Detrimental effects have being described especially since an over consumption of caffeine has being noted. This review focuses on the neurophysiological impact of caffeine and its biochemical pathways in the human body.

Keywords: adenosine receptors; caffeine; energy drinks; neurophysiology.

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Figures

Figure 1
Figure 1
Chemical structure of caffeine (Deng et al., 2008).
Figure 2
Figure 2
Non-degraded stimulus.
Figure 3
Figure 3
Degraded stimulus.
See this image and copyright information in PMC

References

    1. American Psychiatric Association (2000). Diagnostic and Statistical Manual of Mental Disorders: DSM-IV-TR, 4th Edn Washington, DC: American Psychiatric Association
    1. Ashihara H., Sano H., Crozier A. (2008). Caffeine and related purine alkaloids: biosynthesis, catabolism, function and genetic engineering. Phytochemistry 69, 841–856 10.1016/j.phytochem.2007.10.029 - DOI - PubMed
    1. Barry R. J., Clarke A. R., Johnstone S. J., Rushby J. A. (2008). Timing of caffeine’s impact on automatic and central nervous system measures: clarification of arousal effects. Biol. Psychol. 77, 304–316 10.1016/j.biopsycho.2007.11.002 - DOI - PubMed
    1. Barry R. J., Rushby J. A., Wallace M. L., Clarke A. R., Johnstone S. J., Zlojutro I. (2005). Caffeine effects on resting-state arousal. Clin. Neurophysiol. 116, 2693–2700 10.1016/j.clinph.2005.05.003 - DOI - PubMed
    1. Biggs S. N., Smith A., Dorrian J., Reid K., Dawson D., Van den Heuvel C., Baulk S. (2007). Perception of stimulated driving performance after sleep restriction and caffeine. J. Psychosom. Res. 63, 573–577 10.1016/j.jpsychores.2007.06.017 - DOI - PubMed

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