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Editorial
. 2011 Dec;6(12):2735-7.
doi: 10.2215/CJN.10711011.

FGF23 beyond mineral metabolism: a bridge to cardiovascular disease

Tobias E Larsson
Editorial

FGF23 beyond mineral metabolism: a bridge to cardiovascular disease

Tobias E Larsson. Clin J Am Soc Nephrol. 2011 Dec.
No abstract available

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Comment on

  • Fibroblast growth factor 23 in patients undergoing peritoneal dialysis.
    Isakova T, Xie H, Barchi-Chung A, Vargas G, Sowden N, Houston J, Wahl P, Lundquist A, Epstein M, Smith K, Contreras G, Ortega L, Lenz O, Briones P, Egbert P, Ikizler TA, Jueppner H, Wolf M. Isakova T, et al. Clin J Am Soc Nephrol. 2011 Nov;6(11):2688-95. doi: 10.2215/CJN.04290511. Epub 2011 Sep 8. Clin J Am Soc Nephrol. 2011. PMID: 21903990 Free PMC article.

References

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    1. Bai X, Miao D, Li J, Goltzman D, Karaplis AC: Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disorders. Endocrinology 145: 5269–5279, 2004 - PubMed
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    1. ADHR Consortium: Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23. Nat Genet 26: 345–348, 2000 - PubMed

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