Association of cardiovascular and metabolic disease genes with psoriasis

Affiliations

¹ Department of Dermatology, University of California, San Francisco, San Francisco, California, USA.
² Unit of Dermatology and Venereology, Department of Medicine, Karolinska Institutet and Karolinska University Hospital, Stockholm, Sweden.
³ Human Genetics, Genome Institute of Singapore, Singapore, Singapore.
⁴ Division of Human Genetics, Department of Genetics, Washington University School of Medicine, St Louis, Missouri, USA.
⁵ Human Genetics, Genome Institute of Singapore, Singapore, Singapore; Institute for Human Genetics and Department of Laboratory Medicine, University of California, San Francisco, California, USA.
⁶ Department of Dermatology, University of California, San Francisco, San Francisco, California, USA. Electronic address: liaowi@derm.ucsf.edu.

Yingchang Lu et al. J Invest Dermatol. 2013 Mar.

. 2013 Mar;133(3):836-839.

doi: 10.1038/jid.2012.366. Epub 2012 Nov 29.

¹ Department of Dermatology, University of California, San Francisco, San Francisco, California, USA.
² Unit of Dermatology and Venereology, Department of Medicine, Karolinska Institutet and Karolinska University Hospital, Stockholm, Sweden.
³ Human Genetics, Genome Institute of Singapore, Singapore, Singapore.
⁴ Division of Human Genetics, Department of Genetics, Washington University School of Medicine, St Louis, Missouri, USA.
⁵ Human Genetics, Genome Institute of Singapore, Singapore, Singapore; Institute for Human Genetics and Department of Laboratory Medicine, University of California, San Francisco, California, USA.
⁶ Department of Dermatology, University of California, San Francisco, San Francisco, California, USA. Electronic address: liaowi@derm.ucsf.edu.

No abstract available

Conflict of interest statement

Conflict of interest:

The authors state no conflict of interest.

1. Carlsson B, Kindberg E, Buesa J, Rydell GE, Lidon MF, Montava R, et al. The G428A nonsense mutation in FUT2 provides strong but not absolute protection against symptomatic GII.4 Norovirus infection. PLoS One. 2009;4:e5593. - PMC - PubMed
1. Chen H, Hayashi G, Lai OY, Dilthey A, Kuebler PJ, Wong TV, et al. Psoriasis Patients Are Enriched for Genetic Variants That Protect against HIV-1 Disease. Plos Genet. 2012;8:e1002514. - PMC - PubMed
1. Chen H, Poon A, Yeung C, Helms C, Pons J, Bowcock AM, et al. A genetic risk score combining ten psoriasis risk loci improves disease prediction. PLoS One. 2011;6:e19454. - PMC - PubMed
1. Davidovici BB, Sattar N, Prinz JC, Jorg PC, Puig L, Emery P, et al. Psoriasis and systemic inflammatory diseases and potential mechanistic links between skin disease and co-morbid conditions. J Invest Dermatol. 2010;130:1785–1796. - PubMed
1. Devalliere J, Charreau B. The adaptor Lnk (SH2B3): an emerging regulator in vascular cells and a link between immune and inflammatory signaling. Biochemical pharmacology. 2011;82:1391–402. - PubMed