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Case Reports
. 2013 Mar 1;70(3):410-1.
doi: 10.1001/jamaneurol.2013.605.

In vivo demonstration of homonymous hemimacular loss of retinal ganglion cells due to a thalamic lesion using optical coherence tomography

Jiwon Oh  1 , Elias S Sotirchos, Shiv Saidha, Mohamed Ibrahim, Yasir Sepah, Quan Dong Nguyen, Peter A Calabresi
Affiliations
Case Reports

In vivo demonstration of homonymous hemimacular loss of retinal ganglion cells due to a thalamic lesion using optical coherence tomography

Jiwon Oh et al. JAMA Neurol. .
No abstract available

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Figures

Figure 1
Figure 1
A, Axial fluid-attenuated inversion recovery sequence on magnetic resonance imaging showing a hyperintense lesion (arrow) in the right lateral thalamus. The T1-weighted sequence before (B) and after (C) the administration of gadolinium contrast medium shows corresponding subtle contrast enhancement within the lesion (arrows).
Figure 2
Figure 2
A and B, Scanning laser ophthalmoscope microperimetry map (OPKO Health Inc) demonstrating a homonymous hemianopic visual field deficit. The color-coded visual attenuation scale provides the mean retinal sensitivity to a standardized visual stimulus (range, 0–20 dB; with 0 representing the absence of retinal sensitivity and 20 representing maximum retinal sensitivity). C and D, Cirrus high-definition–optical coherence tomographic macular thickness maps (in units of micrometers) demonstrate hemimacular thinning, which corresponds to the demonstrated visual field deficit (green = 5%–95%, yellow = 1%–5%, and red = less than 1% for age-matched controls).
See this image and copyright information in PMC

References

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