The authors reply
- PMID: 23728009
- PMCID: PMC3885351
- DOI: 10.1038/ki.2013.40
The authors reply
Comment on
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Podocyte detachment and reduced glomerular capillary endothelial fenestration promote kidney disease in type 2 diabetic nephropathy.Kidney Int. 2012 Nov;82(9):1010-7. doi: 10.1038/ki.2012.234. Epub 2012 Jun 20. Kidney Int. 2012. PMID: 22718189 Free PMC article. Clinical Trial.
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Is endothelial dysfunction more deleterious than podocyte injury in diabetic nephropathy?Kidney Int. 2013 Jun;83(6):1202-3. doi: 10.1038/ki.2013.36. Kidney Int. 2013. PMID: 23728007 No abstract available.
References
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- Nakagawa T. Is endothelial dysfunction more deleterious than podocyte injury in diabetic nephropathy? Kidney Int. 2013;83:1202–1203. - PubMed
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- Lemley KV, Blouch K, Abdullah I, et al. Glomerular permselectivity at the onset of nephropathy in type 2 diabetes mellitus. J Am Soc Nephrol. 2000;11:2095–2105. - PubMed
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- Salmon AH, Satchell SC. Endothelial glycocalyx dysfunction in disease: albuminuria and increased microvascular permeability. J Pathol. 2012;226:562–74. - PubMed
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- Nakagawa T, Sato W, Glushakova O, et al. Diabetic endothelial nitric oxide synthase knockout mice develop advanced diabetic nephropathy. J Am Soc Nephrol. 2007;18:539–550. - PubMed
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