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. 2012 Oct;5(5):456-458.
doi: 10.1093/ckj/sfs102.

Contrast-induced acute kidney injury following iodine opacification other than by intravascular injection

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Contrast-induced acute kidney injury following iodine opacification other than by intravascular injection

Tilman Perrin et al. Clin Kidney J. 2012 Oct.

Abstract

Contrast-induced acute kidney injury (CI-AKI) classically occurs following the intravascular administration of iodinated contrast medium (CM). However, some cases of iodine-induced nephrotoxicity have been reported in patients who did not receive intravascular CM, as a consequence of iodine absorption through mucosae, burned skin or interstitial tissues. Recently, we observed the first case of CI-AKI occurring after an enteroclysis without any direct intravascular injection of CM. Here, we report this case, and review other clinical situations in which renal toxicity has been reported following the non-intravascular use of iodinated compounds.

Keywords: acute kidney injury; acute renal failure; contrast-induced acute kidney injury; enteroclysis; iodine toxicity.

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Figures

Fig. 1.
Fig. 1.
Evolution of blood creatinine level after enteroclysis (arrow).
Fig. 2.
Fig. 2.
Abdominal native CT-scan made 1 h after enteroclysis showing contrast media in the enterostomy (A), intestine (B) and renal pelvis bilaterally (C).

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