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Editorial
. 2014 Feb;5(1):1-2.
doi: 10.1007/s12975-014-0324-3. Epub 2014 Jan 27.

Dysregulation of diverse ion transport pathways controlling cell volume homoestasis contribute to neuroglial cell injury following ischemic stroke

Dandan Sun  1 , Kristopher T Kahle
Affiliations
Editorial

Dysregulation of diverse ion transport pathways controlling cell volume homoestasis contribute to neuroglial cell injury following ischemic stroke

Dandan Sun et al. Transl Stroke Res. 2014 Feb.
No abstract available

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Conflict of interest statement

The authors declare no conflict of interest.

References

    1. Brian P Walcott MD. Disruption of ion homeostasis in the neurogliovascular unit underlies the pathogenesis of ischemic cerebral edema-TRSR329R. doi:10.1007/s12975-013-0307-9 - PMC - PubMed
    1. Shan Ping Yu MD PhD. Ionic regulation of cell volume changes and the phenotype of cell death after ischemic stroke-TRSR355R1. doi:10.1007/s12975-013-0314-x - PMC - PubMed
    1. Michael Norenberg. Sulfonylurea receptor 1 contributes to the astrocyte swelling and brain edema in acute liver failure-TRSR346. doi:10.1007/s12975-014-0328-z - PMC - PubMed
    1. Niyathi Hegde Shah. Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration-TRSR334R1. doi:10.1007/s12975-013-0297-7 - PMC - PubMed
    1. Zhi-Gang Xiong. Translational strategies for neuroprotection in ischemic stroke—focusing on acid sensing ion channel 1a-TRSR344R1. doi:10.1007/s12975-013-0319-5 - PMC - PubMed

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