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Editorial
. 2014 Feb 13;5(2):e1062.
doi: 10.1038/cddis.2014.50.

The 1000th publication features protection of normal cells during chemotherapy: a land mark for Cell Death & Disease

Y Shi  1
Affiliations
Editorial

The 1000th publication features protection of normal cells during chemotherapy: a land mark for Cell Death & Disease

Y Shi. Cell Death Dis. .
No abstract available

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Figures

Figure 1
Figure 1
The chemoprotective ability of rocaglamide (ROC-A). It is demonstrated that the major mechanism of ROC-A protection against DNA-damage-induced cell death is dependent on its inhibition of p53 expression in nonmalignant cells. As such, in p53 deficiency or mutation cancer cells, ROC-A cannot exert such chemoprotective effects
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References

    1. Becker MS, Schmezer P, Breuer R, Haas SF, Essers MA, Krammer PH, et al. The traditional Chinese medical compound Rocaglamide protects nonmalignant primary cells from DNA damage-induced toxicity by inhibition of p53 expression. Cell Death Dis. 2014;5:e1000. - PMC - PubMed

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