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Review
. 2014 Jun;20(3 Neurology of Systemic Disease):580-97.
doi: 10.1212/01.CON.0000450967.76452.f2.

Neurologic complications of bariatric surgery

Review

Neurologic complications of bariatric surgery

Neeraj Kumar. Continuum (Minneap Minn). 2014 Jun.

Abstract

Purpose of review: The increasing utilization of bariatric surgery has been accompanied by an increased incidence and awareness of related neurologic complications. The purpose of this review is to provide up-to-date information on the neurologic complications related to bariatric surgery.

Recent findings: Neurologic complications related to bariatric surgery are predominantly due to nutrient deficiencies. Common early complications include Wernicke encephalopathy due to thiamine deficiency, and late complications include myelopathy or myeloneuropathy due to vitamin B12 or copper deficiency.

Summary: Early recognition and prompt institution of treatment is essential to prevent long-term disability. Often, life-long supplementation may be required.

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Figures

Figure 4-1.
Figure 4-1.
Axial fluid-attenuated inversion recovery (FLAIR) brain MRI of a patient with Wernicke encephalopathy showing bilateral, symmetric, hyperintense signal involving the medial thalami (arrows). This patient also had signal change in the periaqueductal gray matter (not shown). This patient’s Wernicke encephalopathy occurred weeks after bariatric surgery. Reprinted with permission from Foster D, et al, Neurology. © 2005, American Academy of Neurology. www.neurology.org/content/65/12/1987.short?sid=3f566b80-7408-416b-988e-edef3b1bf70d.
Figure 4-2.
Figure 4-2.
Sagittal (A) and axial (B) cervical spine MRI from a patient with copper deficiency myelopathy showing a diffuse cord signal abnormality (arrows) involving the posterior aspect of the cord (dorsal column). Similar MRI findings are seen in vitamin B12 deficiency–associated myelopathy.

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