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. 2014;7(2):199-218.
doi: 10.3400/avd.pad.14.01000.

5(th) Asian PAD Workshop

No authors listed

5(th) Asian PAD Workshop

No authors listed. Ann Vasc Dis. 2014.
No abstract available

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Figures

Fig. 1
Fig. 1
Tokyo tower was illuminated green on Aged Day in Japan
Fig. 2
Fig. 2
Dual effect of beraprost in PAD
Fig. 1
Fig. 1
Prognosis of CKD by GFR and albuminuria categories (KIDGO 2012)2)
Fig. 2
Fig. 2
Association between eGFR levels and prevalence of PAD8)
Fig. 3
Fig. 3
Incremental correlation between prevalence of abnormal ABI and stage of CKD
Fig. 4
Fig. 4
Relationship of high and low ABI to all-cause and cardiovascular disease mortality in the Strong Heart Study
Fig. 5
Fig. 5
Suggested patient management recommendations
Fig. 6
Fig. 6
PAD patient with concurrent Type 2 DM on insulin therapy (duration of diabetes: 24 years, 65-year-old male) before and after beraprost 120μg/day15)
Fig. 1
Fig. 1
Target cells of PGs2)
Fig. 2
Fig. 2
Effects of beraprost on PAD symptoms: coldness of limbs and numbness5)
Fig. 3
Fig. 3
Effects of beraprost on blood flow in lower limbs: change of ABI5)
Fig. 4
Fig. 4
Improvement of pericutaneous PaO2 with beraprost6)
Fig. 5
Fig. 5
Inhibition of the release of PDGF in vitro8)
Fig. 6
Fig. 6
Proposed action of LDLA13)
Fig. 1
Fig. 1
Causes of ESRD4)
Fig. 2
Fig. 2
Interaction of CKD-MBD in ESRD patients6,7)
Fig. 3
Fig. 3
Mechanisms of vascular calcification in CKD6,7) MGP, matrix GLA protein; OPG, osteoprotegerin
Fig. 4
Fig. 4
Major cause of death5)
Fig. 5
Fig. 5
Hypothetical role of mild to moderate CKD in the occurrence of cardiovascular dysfunction8)
Fig. 6
Fig. 6
Risk of PAD in CKD patients in the ARIC study12)
Fig. 7
Fig. 7
Mortality rate in CKD/PAD patients14)
Fig. 8
Fig. 8
Urine protein excretion and glomerular volume in OLETF rats following beraprost treatment15)
Fig. 1
Fig. 1
Vascular risk in patients with microalbuminuria2)
Fig. 2
Fig. 2
Cardiovascular survival according to the absence vs the presence of microalbuminuria (b) and PAD (b)3)
Fig. 3
Fig. 3
Kaplan-Meier curve for remaining free of PAD by level of eGFR6)
Fig. 4
Fig. 4
Relative risks of future CV events7)
Fig. 5
Fig. 5
Risk of PAD by disease and CRP category8)
Fig. 1
Fig. 1
Percentage of antiplatelet drug-treated patients having ABI <0.914)
Fig. 2
Fig. 2
Changes in SPP. (A) SPP in instep, (B) SPP in sole. ■ indicate group A (beraprost, n=35) and ■ indicate group B (cilostazol or sarpogrelate, n=33).15) *p<0.05, #p<0.01 vs pretreatment values. NS, not significant
Fig. 3
Fig. 3
Effect of treatment on heart rate. ● indicate heart rate of group A (beraprost, n=35), ■ indicate heart rate of cilostazol-treated patients in group B (n=18).15) #p<0.01 vs pretreatment values
Fig. 4
Fig. 4
Pooled survival curves (meta-analysis)17)
See this image and copyright information in PMC

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