Tryptophan catabolism is unaffected in chronic granulomatous disease

Ghassan J Maghzal¹, Susann Winter², Bettina Wurzer³, Beng H Chong⁴, Rikard Holmdahl², Roland Stocker¹

Affiliations

¹ 1] Vascular Biology Division, Victor Chang Cardiac Research Institute, Darlinghurst, New South Wales 2010, Australia [2] School of Medicine, University of New South Wales, New South Wales 2052, Australia [3] School of Medical Sciences and Bosch Institute, The University of Sydney, Camperdown, New South Wales 2050, Australia.
² Department of Medical Biochemistry and Biophysics, Karolinska Institutet, 171 77 Stockholm, Sweden.
³ School of Medical Sciences and Bosch Institute, The University of Sydney, Camperdown, New South Wales 2050, Australia.
⁴ Centre for Vascular Research, University of New South Wales, New South Wales 2052, Australia.

PMID: 25341792
DOI: 10.1038/nature13844

Comment

Tryptophan catabolism is unaffected in chronic granulomatous disease

Ghassan J Maghzal et al. Nature. 2014.

. 2014 Oct 23;514(7523):E16-7.

doi: 10.1038/nature13844.

Authors

Ghassan J Maghzal¹, Susann Winter², Bettina Wurzer³, Beng H Chong⁴, Rikard Holmdahl², Roland Stocker¹

Affiliations

¹ 1] Vascular Biology Division, Victor Chang Cardiac Research Institute, Darlinghurst, New South Wales 2010, Australia [2] School of Medicine, University of New South Wales, New South Wales 2052, Australia [3] School of Medical Sciences and Bosch Institute, The University of Sydney, Camperdown, New South Wales 2050, Australia.
² Department of Medical Biochemistry and Biophysics, Karolinska Institutet, 171 77 Stockholm, Sweden.
³ School of Medical Sciences and Bosch Institute, The University of Sydney, Camperdown, New South Wales 2050, Australia.
⁴ Centre for Vascular Research, University of New South Wales, New South Wales 2052, Australia.

PMID: 25341792
DOI: 10.1038/nature13844

No abstract available

PubMed Disclaimer

Comment in

Romani & Puccetti reply.
Romani L, Puccetti P. Romani L, et al. Nature. 2014 Oct 23;514(7523):E18. doi: 10.1038/nature13845. Nature. 2014. PMID: 25341793 No abstract available.

Comment on

Defective tryptophan catabolism underlies inflammation in mouse chronic granulomatous disease.
Romani L, Fallarino F, De Luca A, Montagnoli C, D'Angelo C, Zelante T, Vacca C, Bistoni F, Fioretti MC, Grohmann U, Segal BH, Puccetti P. Romani L, et al. Nature. 2008 Jan 10;451(7175):211-5. doi: 10.1038/nature06471. Nature. 2008. PMID: 18185592

References

1. Medicine (Baltimore). 2000 May;79(3):170-200 - PubMed
1. J Biol Chem. 1994 May 20;269(20):14457-64 - PubMed
1. Nature. 2008 Jan 10;451(7175):211-5 - PubMed
1. FEBS Lett. 1982 Aug 2;144(2):199-203 - PubMed
1. J Exp Med. 1997 Jan 20;185(2):207-18 - PubMed

Publication types

Actions
Actions

MeSH terms

Actions
Actions
Actions
Actions
Actions
Actions

Substances

Actions
Actions

LinkOut - more resources

Full Text Sources
- Nature Publishing Group
- Ovid Technologies, Inc.
Other Literature Sources
- scite Smart Citations

Save citation to file

Email citation

Add to Collections

Add to My Bibliography

Your saved search

Create a file for external citation management software

Your RSS Feed

Tryptophan catabolism is unaffected in chronic granulomatous disease

Affiliations

Tryptophan catabolism is unaffected in chronic granulomatous disease

Authors

Affiliations

Comment in

Comment on

References

Publication types

MeSH terms

Substances

LinkOut - more resources

Full Text Sources

Other Literature Sources