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. 2012 Sep 6;2012(1):5.
doi: 10.5339/gcsp.2012.5. eCollection 2012.

Hypertrophic cardiomyopathy: The first century 1869-1969

Eugene Braunwald  1
Affiliations

Hypertrophic cardiomyopathy: The first century 1869-1969

Eugene Braunwald. Glob Cardiol Sci Pract. .
No abstract available

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Figures

Figure 1.
Figure 1.. Gross anatomy of the heart in a patient with familial HCM (Ref. [6]).
Figure 2.
Figure 2.. Left heart dynamics in three siblings with obstructive HCM. LV = left ventricular (Ref. [9]).
Figure 3.
Figure 3.. Hemodynamic effects of 2 μg isoproterenol. Isuprel = isoproterenol, C.O. = cardiac output, Grad. = gradient, Eff. Orif. = effective orifice (Ref. [10]).
Figure 4.
Figure 4.. Circulatory response to nitroglycerin in patient B.W. with HCM. LV, left ventricular pressure pulse; BA, brachial arterial pressure pulse. (From Braunwald E, Oldham H N Jr, Ross J Jr, et al. The circulatory response of patients with idiopathic hypertrophic subaortic stenosis to nitroglycerin and to the Valsalva maneuver. Circulation 1964;29:422–431).
See this image and copyright information in PMC

References

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    1. Schmincke A. Ueber linkseitige muskulose conustenosen. Deutsche Med Wochenschr. 1907;33:2082–2085.
    1. Levy RL, von Glahn WC. Cardiac hypertrophy of unknown cause. A study of the clinical and pathologic features in ten adults. Am Heart J. 1944;28:714–741.
    1. Evans W. Familial cardiomegaly. Br Heart J. 1949;11:68–82. - PMC - PubMed
    1. Paré JAP, Fraser RG, Pirozynski WJ, Shanks JA, Stubington D. Hereditary cardiovascular dysplasia. A form of familial cardiomyopathy. Am J Med. 1961;31:37–62. - PubMed