Diabetic cardiomyopathy: catabolism driving metabolism

Affiliations

¹ From Departments of Internal Medicine (Cardiology) (Z.V.W., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas.
² From Departments of Internal Medicine (Cardiology) (Z.V.W., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas. joseph.hill@utsouthwestern.edu.

Editorial

Zhao V Wang et al. Circulation. 2015.

. 2015 Mar 3;131(9):771-3.

doi: 10.1161/CIRCULATIONAHA.115.015357. Epub 2015 Jan 30.

¹ From Departments of Internal Medicine (Cardiology) (Z.V.W., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas.
² From Departments of Internal Medicine (Cardiology) (Z.V.W., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas. joseph.hill@utsouthwestern.edu.

No abstract available

Keywords: Editorials; diabetic cardiomyopathies; metabolism.

Comment on

Upregulation of MG53 induces diabetic cardiomyopathy through transcriptional activation of peroxisome proliferation-activated receptor α.
Liu F, Song R, Feng Y, Guo J, Chen Y, Zhang Y, Chen T, Wang Y, Huang Y, Li CY, Cao C, Zhang Y, Hu X, Xiao RP. Liu F, et al. Circulation. 2015 Mar 3;131(9):795-804. doi: 10.1161/CIRCULATIONAHA.114.012285. Epub 2015 Jan 30. Circulation. 2015. PMID: 25637627

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