doi: 10.1016/s0140-6736(89)92752-9.
Campylobacter pylori and duodenal ulcers: the gastrin link
Affiliations
- PMID: 2566737
- DOI: 10.1016/s0140-6736(89)92752-9
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Campylobacter pylori and duodenal ulcers: the gastrin link
Lancet.
.
Abstract
The possibility that Campylobacter pylori (CP) in the gastric antrum stimulates gastrin release in duodenal ulcer (DU) disease was examined in 31 patients. The 25 patients with antral colonisation with CP had higher basal and meal-stimulated plasma gastrin concentrations, and higher peak acid output (PAO), than did the 6 without CP in the autumn.
Comment in
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Campylobacter pylori and acid secretion.Lancet. 1989 Jul 22;2(8656):212-3. Lancet. 1989. PMID: 2568533 No abstract available.
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Campylobacter pylori, gastrin, and duodenal ulcer.Lancet. 1989 Aug 12;2(8659):389. doi: 10.1016/s0140-6736(89)90568-0. Lancet. 1989. PMID: 2569578 No abstract available.
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Omeprazole, campylobacter pylori, and duodenal ulcer.Lancet. 1989 Aug 12;2(8659):389-90. doi: 10.1016/s0140-6736(89)90569-2. Lancet. 1989. PMID: 2569579 No abstract available.
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Lowered gastrin and gastric acidity after eradication of Campylobacter pylori in duodenal ulcer.Lancet. 1989 Aug 26;2(8661):499-500. doi: 10.1016/s0140-6736(89)92105-3. Lancet. 1989. PMID: 2570202 No abstract available.
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Campylobacter pylori and acid secretion.Lancet. 1989 Sep 2;2(8662):562. doi: 10.1016/s0140-6736(89)90684-3. Lancet. 1989. PMID: 2570259 No abstract available.
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