Introduction

Richard E Frye^{1

2}, John Slattery^{1

2}

Affiliations

¹ Autism Research Program, Arkansas Children's Hospital Research Institute, Little Rock, AR, USA.
² Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, AR, USA.

Richard E Frye et al. Microb Ecol Health Dis. 2015.

. 2015 May 7:26:28168.

doi: 10.3402/mehd.v26.28168. eCollection 2015.

Richard E Frye^{1

2}, John Slattery^{1

2}

¹ Autism Research Program, Arkansas Children's Hospital Research Institute, Little Rock, AR, USA.
² Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, AR, USA.

No abstract available

References

1. Developmental Disabilities Monitoring Network Surveillance Year Principal I, Centers for Disease C, Prevention. Prevalence of autism spectrum disorder among children aged 8 years – autism and developmental disabilities monitoring network, 11 sites, United States, 2010. MMWR Surveill Summ. 2014;63:1–21. - PubMed
1. Rossignol DA, Frye RE. A review of research trends in physiological abnormalities in autism spectrum disorders: immune dysregulation, inflammation, oxidative stress, mitochondrial dysfunction and environmental toxicant exposures. Mol Psychiatry. 2012;17:389–401. - PMC - PubMed
1. Schaefer GB, Mendelsohn NJ, Professional P, Guidelines C. Clinical genetics evaluation in identifying the etiology of autism spectrum disorders: 2013 guideline revisions. Genet Med. 2013;15:399–407. - PubMed
1. Frye RE, James SJ. Metabolic pathology of autism in relation to redox metabolism. Biomark Med. 2014;8:321–30. - PubMed
1. Rossignol DA, Frye RE. Evidence linking oxidative stress, mitochondrial dysfunction, and inflammation in the brain of individuals with autism. Front Physiol. 2014;5:150. - PMC - PubMed