After all, plasmalemmal expression of type-1 VDAC can be understood. Phosphorylation, nitrosylation, and channel modulators work together in vertebrate cell volume regulation and either apoptotic pathway
- PMID: 25964761
- PMCID: PMC4410597
- DOI: 10.3389/fphys.2015.00126
After all, plasmalemmal expression of type-1 VDAC can be understood. Phosphorylation, nitrosylation, and channel modulators work together in vertebrate cell volume regulation and either apoptotic pathway
Keywords: Alzheimer's Disease; IClswell; VRAC; VSOAC; autism; cystic fibrosis; malaria; porin.
Figures

Similar articles
-
Phosphorylation, nitrosation and plasminogen K3 modulation make VDAC-1 lucid as part of the extrinsic apoptotic pathway-Resulting thesis: Native VDAC-1 indispensible for finalisation of its 3D structure.Biochim Biophys Acta. 2015 Jun;1848(6):1410-6. doi: 10.1016/j.bbamem.2015.02.031. Epub 2015 Mar 11. Biochim Biophys Acta. 2015. PMID: 25771449 Review.
-
New findings concerning vertebrate porin II--on the relevance of glycine motifs of type-1 VDAC.Mol Genet Metab. 2013 Apr;108(4):212-24. doi: 10.1016/j.ymgme.2013.01.008. Epub 2013 Jan 26. Mol Genet Metab. 2013. PMID: 23419876 Review.
-
Opening up of plasmalemma type-1 VDAC to form apoptotic "find me signal" pathways is essential in early apoptosis - evidence from the pathogenesis of cystic fibrosis resulting from failure of apoptotic cell clearance followed by sterile inflammation.Mol Genet Metab. 2014 Apr;111(4):439-44. doi: 10.1016/j.ymgme.2014.02.001. Epub 2014 Feb 13. Mol Genet Metab. 2014. PMID: 24613483 Review.
-
Studies on human porin XXI: gadolinium opens Up cell membrane standing porin channels making way for the osmolytes chloride or taurine-A putative approach to activate the alternate chloride channel in cystic fibrosis.Mol Genet Metab. 2000 Mar;69(3):240-51. doi: 10.1006/mgme.2000.2968. Mol Genet Metab. 2000. PMID: 10767179
-
Human voltage-dependent anion-selective channel expressed in the plasmalemma of Xenopus laevis oocytes.Int J Biochem Cell Biol. 2000 Oct;32(10):1075-84. doi: 10.1016/s1357-2725(00)00047-9. Int J Biochem Cell Biol. 2000. PMID: 11091140
Cited by
-
Pharmacological modulation of mitochondrial ion channels.Br J Pharmacol. 2019 Nov;176(22):4258-4283. doi: 10.1111/bph.14544. Epub 2019 Jan 2. Br J Pharmacol. 2019. PMID: 30440086 Free PMC article. Review.
-
Neuronal ER-Signalosome Proteins as Early Biomarkers in Prodromal Alzheimer's Disease Independent of Amyloid-β Production and Tau Phosphorylation.Front Mol Neurosci. 2022 May 5;15:879146. doi: 10.3389/fnmol.2022.879146. eCollection 2022. Front Mol Neurosci. 2022. PMID: 35600079 Free PMC article.
-
The Critical Period for Neuroprotection by Estrogen Replacement Therapy and the Potential Underlying Mechanisms.Curr Neuropharmacol. 2020;18(6):485-500. doi: 10.2174/1570159X18666200123165652. Curr Neuropharmacol. 2020. PMID: 31976839 Free PMC article. Review.
-
Preserving Insulin Secretion in Diabetes by Inhibiting VDAC1 Overexpression and Surface Translocation in β Cells.Cell Metab. 2019 Jan 8;29(1):64-77.e6. doi: 10.1016/j.cmet.2018.09.008. Epub 2018 Oct 4. Cell Metab. 2019. PMID: 30293774 Free PMC article.
-
Estrogen Interactions With Lipid Rafts Related to Neuroprotection. Impact of Brain Ageing and Menopause.Front Neurosci. 2018 Mar 6;12:128. doi: 10.3389/fnins.2018.00128. eCollection 2018. Front Neurosci. 2018. PMID: 29559883 Free PMC article. Review.
References
-
- Benz R., Maier E., Thinnes F. P., Götz H., Hilschmann N. (1992). Studies on human porin. VII. The channel properties of the human B-lymphocyte membrane-derived “Porin 31HL” are similar to those of mitochondrial porins. Biol. Chem. Hoppe Seyler 373, 295–303. 10.1515/bchm3.1992.373.1.295 - DOI - PubMed
LinkOut - more resources
Full Text Sources
Other Literature Sources