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Review
. 2015 Nov;66(5):869-83.
doi: 10.1053/j.ajkd.2015.04.042. Epub 2015 Jun 6.

Onco-nephrology: Core Curriculum 2015

Eric P Cohen  1 , Jean-Marie Krzesinski  2 , Vincent Launay-Vacher  3 , Ben Sprangers  4
Affiliations
Review

Onco-nephrology: Core Curriculum 2015

Eric P Cohen et al. Am J Kidney Dis. 2015 Nov.
No abstract available

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Conflict of interest statement

Financial disclosure: The authors declare that they have no relevant financial interests.

Figures

Figure 1
Figure 1
Causes of acute kidney injury in critically ill cancer patients. Data from Soares et al (Prognosis of critically ill patients with cancer and acute renal dysfunction. J Clin Oncol. 2006;24:4003–4010).
Figure 2
Figure 2
Paraprotein-related kidney injury. A growing number of kidney diseases have been associated with paraproteinemia. Abbreviations: GN, glomerulonephritis; Ig, immunoglobulin; MCD minimal change disease; MIDD, monoclonal immunoglobulin deposition disease; PGNMID, proliferative glomerulonephritis with monoclonal immune deposits; TMA, thrombotic microangiopathy.
Figure 3
Figure 3
The occurrence of acute kidney injury after HSC transplantation. Acute kidney injury (AKI) is more common after myeloablative HSC transplantation, and more often leads to a requirement for dialysis. AKI is defined here as a more-than-doubling of the serum creatinine. Data from Parikh et al (Comparison of ARF After Myeloablative and Nonmyeloablative Hematopoietic Cell Transplantation. Am J Kidney Dis. 2005;45:502–509).
Figure 4
Figure 4
The prevalence of CKD after HSC transplantation in children and adults, based on studies from 2007 onward. The arithmetic average for all studies is 13%. Reproduced from Cohen et al (Chronic Kidney Disease After Hematopoietic Stem Cell Transplantation. Semin Nephrol. 2010;30:627–634) with permission from Elsevier.
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