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Editorial
. 2015 Sep;121(3):594-596.
doi: 10.1213/ANE.0000000000000871.

HCN1 Channels as Targets for Volatile Anesthetics: Coming to the Fore

Peter A Goldstein  1
Affiliations
Editorial

HCN1 Channels as Targets for Volatile Anesthetics: Coming to the Fore

Peter A Goldstein. Anesth Analg. 2015 Sep.
No abstract available

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References

    1. Franks NP, Lieb WR. Molecular and cellular mechanisms of general anaesthesia. Nature. 1994;367:607–14
    1. Tokimasa T, Sugiyama K, Akasu T, Muteki T. Volatile anaesthetics inhibit a cyclic AMP-dependent sodium-potassium current in cultured sensory neurones of bullfrog. Br J Pharmacol. 1990;101:190–2
    1. Sirois JE, Pancrazio JJ, Lynch C III, Bayliss DA. Multiple ionic mechanisms mediate inhibition of rat motoneurones by inhalation anaesthetics. J Physiol. 1998;512(Pt 3):851–62
    1. Sirois JE, Lynch C III, Bayliss DA. Convergent and reciprocal modulation of a leak K+ current and I(h) by an inhalational anaesthetic and neurotransmitters in rat brainstem motoneurones. J Physiol. 2002;541:717–29
    1. Chen X, Sirois JE, Lei Q, Talley EM, Lynch C III, Bayliss DA. HCN subunit-specific and cAMP-modulated effects of anesthetics on neuronal pacemaker currents. J Neurosci. 2005;25:5803–14

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