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Comment
. 2016 Feb;63(2):366.
doi: 10.1002/pbc.25798. Epub 2015 Oct 21.

Reply to: Glucarpidase for the Treatment of Methotrexate-Induced Renal Dysfunction and Delayed Methotrexate Excretion

Brigitte C Widemann  1
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Comment

Reply to: Glucarpidase for the Treatment of Methotrexate-Induced Renal Dysfunction and Delayed Methotrexate Excretion

Brigitte C Widemann. Pediatr Blood Cancer. 2016 Feb.
No abstract available

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Conflict of interest statement

Conflict of interest: Nothing to declare.

Comment on

References

    1. Meyers PA. Glucarpidase for the treatment of methotrexate-induced renal dysfunction and delayed methotrexate excretion. Pediatr Blood Cancer 2015; doi:10.1002/pbc.25748. - DOI - PubMed
    1. Meyers PA, Flombaum C. High-dose methotrexate-induced renal dysfunction: Is glucarpidase necessary for rescue? J Clin Oncol 2011;29:e180; author reply e181. - PubMed
    1. Widemann BC, Adamson PC. Understanding and managing methotrexate nephrotoxicity. Oncologist 2006;11:694–703. - PubMed
    1. Widemann BC, Balis FM, Kim A, Boron M, Jayaprakash N, Shalabi A, O’Brien M, Eby M, Cole DE, Murphy RF, Fox E, Ivy P, Adamson PC. Glucarpidase, leucovorin, and thymidine for high-dose methotrexate-induced renal dysfunction: Clinical and pharmacologic factors affecting outcome. J Clin Oncol 2010;28:3979–3986. - PMC - PubMed

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