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Case Reports
. 2016 Winter;9(1):64-7.

Endoscopic pyloroplasty for severe gastric outlet obstruction due to alkali ingestion in a child

Seyed Mohsen Dehghani  1 , Mitra Aldaghi  2 , Hazhir Javaherizadeh  3
Affiliations
Case Reports

Endoscopic pyloroplasty for severe gastric outlet obstruction due to alkali ingestion in a child

Seyed Mohsen Dehghani et al. Gastroenterol Hepatol Bed Bench. 2016 Winter.

Abstract

A common belief is that alkali ingestion causes severe esophageal damage and limited gastric injury due to the buffering action of acid. Gastric injury has been observed in patients who ingested alkali. Gastric outlet obstruction (GOO) secondary to caustic ingestion occurs due to fibrosis after resolution of the acute injury and inflammation, most commonly 6 to 12 weeks after initial ingestion. The traditional treatment for GOO related to ingestion of corrosive agents is surgery. Experience with endoscopic balloon dilation of corrosive-induced GOO is limited in children. This is the first report of endoscopic pyloroplasty in a child with GOO due to caustic alkalis ingestion that was treated with balloon dilation (using TTS balloon ranging from 6-15 mm) in Iran. Four dilation sessions were required for symptomatic relief of dysphagia. After one year of follow up, weight gain was normal.

Keywords: Endoscopic pyloroplasty; Gastric injury; Obstruction.

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Figures

Figure 1
Figure 1
Middle third of Esophagus
Figure 2
Figure 2
Antrum
Figure 3
Figure 3
Pyloric stenosis after one month
Figure 4
Figure 4
Pyloric stenosis after 2 months
Figure 5
Figure 5
Normal pyloric canal and duodenum after one year
See this image and copyright information in PMC

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