Similar articles

• Superoxide dismutase plus catalase therapy delays neither cell death nor the loss of the TTC reaction in experimental myocardial infarction in dogs.

  Tanaka M, Richard VJ, Murry CE, Jennings RB, Reimer KA. Tanaka M, et al. J Mol Cell Cardiol. 1993 Apr;25(4):367-78. doi: 10.1006/jmcc.1993.1043. J Mol Cell Cardiol. 1993. PMID: 8340930
• Free radicals and myocardial injury: pharmacologic implications.

  Werns SW, Shea MJ, Lucchesi BR. Werns SW, et al. Circulation. 1986 Jul;74(1):1-5. doi: 10.1161/01.cir.74.1.1. Circulation. 1986. PMID: 3011308 Review. No abstract available.
• [Prevention of myocardial damage by oxygen free radicals. Theoretical premises and experimental data].

  Boffa GM, Ferrari R, Dalla Volta S. Boffa GM, et al. Cardiologia. 1985 Oct;30(10):931-3. Cardiologia. 1985. PMID: 3939564 Italian. No abstract available.
• Post-anoxic hemodynamic performance. The effect of allopurinol and superoxide dismutase/catalase.

  Bergsland J, LoBalsamo L, Lajos P, Mookerjee B. Bergsland J, et al. Transplant Proc. 1987 Oct;19(5):4165-6. Transplant Proc. 1987. PMID: 3314010 No abstract available.
• Free radicals and myocardial ischemia and reperfusion injury.

  Simpson PJ, Lucchesi BR. Simpson PJ, et al. J Lab Clin Med. 1987 Jul;110(1):13-30. J Lab Clin Med. 1987. PMID: 3298506 Review.