Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
Case Reports
. 2016 Feb 17;6(1):30460.
doi: 10.3402/jchimp.v6.30460. eCollection 2016.

Ventricular septal defect: early against late surgical repair

Affiliations
Case Reports

Ventricular septal defect: early against late surgical repair

Waqas J Siddiqui et al. J Community Hosp Intern Med Perspect. .

Abstract

Ventricular septal defect (VSD) is a rare complication of right ventricular infarction (RVI) which is associated with significant mortality, if not treated appropriately. It typically occurs within the first 10-14 days after myocardial infarction. Surgical repair has been shown to reduce in-hospital mortality from 90% to 33-45%. Early surgical VSD repair has also been associated with high 30-day operative mortality of 34-37%. Furthermore, after an acute MI the friable myocardium enhances the risk of recurrent VSD with early surgical repair. We present a case of a middle-aged woman who developed VSD after an RVI. Her surgical repair was delayed by 2 weeks due to development of Staphylococcus aureus bacteremia. During this period, she was managed medically and later on underwent percutaneous repair with an amplatzer VSD occluder device. Keeping this patient encounter in mind, we would like to emphasize on the limited recommendations available for early against late surgical repair of VSD.

Keywords: early against late repair; right ventricular myocardial infarction; ventricular septal rupture.

PubMed Disclaimer

Figures

Fig. 1
Fig. 1
EKG showing sinus tachycardia with q waves and ST segment elevations of almost 2 mm in leads III and aVF (arrow) along with ST segment depressions in I and aVL leads. This implies that there is possible inferior wall infarction. Left atrial enlargement also noted.
Fig. 2
Fig. 2
Echocardiogram with color Doppler displaying a ventricular septal defect postmyocardial infarction (arrow).
Fig. 3
Fig. 3
Transesophageal echocardiogram showing ventricular septal defect (arrow) with size.
Fig. 4
Fig. 4
3D echocardiogram showing ventricular septal defect (red arrow) and necrosis (black arrow).

References

    1. Manno BV, Bemis CE, Carver J, Mintz GS. Right ventricular infarction complicated by right to left shunt. J Am Coll Cardiol. 1983;1:554–57. - PubMed
    1. Moore CA, Nygaard TW, Kaiser DL, Cooper AA, Gibson RS. Postinfarction ventricular septal rupture: the importance of location of infarction and right ventricular function in determining survival. Circulation. 1986;74:45. - PubMed
    1. Crenshaw BS, Granger CB, Birnbaum Y, Pieper KS, Morris DC, Kleiman NS, et al. Risk factors, angiographic patterns, and outcomes in patients with ventricular septal defect complicating acute myocardial infarction. Circulation. 2000;101:27–32. - PubMed
    1. Coskun KO, Coskun ST, Popov AF, Hinz J, Schmitto JD, Bockhorst K, et al. Experiences with surgical treatment of ventricle septal defect as a post infarction complication. J Cardiothorac Surg. 2009;4:3. - PMC - PubMed
    1. Pang PY, Sin YK, Lim CH, Tan TE, Lim SL, Chao VT, et al. Outcome and survival analysis of surgical repair of post-infarction ventricular septal rupture. J Cardiothorac Surg. 2013;8:44. - PMC - PubMed

Publication types

LinkOut - more resources