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. 2016 Sep 27;113(39):10745-7.
doi: 10.1073/pnas.1612925113. Epub 2016 Sep 20.

Clathrin in plant defense signaling and execution

Viktor Žárský  1
Affiliations

Clathrin in plant defense signaling and execution

Viktor Žárský. Proc Natl Acad Sci U S A. .
No abstract available

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Conflict of interest statement

The author declares no conflict of interest.

Figures

Fig. 1.
Fig. 1.
In the WT (Left), the activated complex of PRK with the BAK1 coreceptor is formed upon binding the pathogen-derived ligand (small violet ellipse), and the clathrin-dependent endocytosis (CCV) is initiated, resulting in the transport of the ligand–receptor complex to the vacuole via a late endosomal (LE) MVB compartment bypassing the Golgi (GA) or TGN/EE compartments. Pathogens might try to block this defense mechanism by injection of effector proteins (PE and dashed violet line). In clathrin mutants (Right), this process is inhibited, and in the case of FLS2-dependent defense, this results in an increased sensitivity to Pseudomonas syringae infection.
See this image and copyright information in PMC

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