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Comment
. 2016 Dec;75(12):e82.
doi: 10.1136/annrheumdis-2016-210529. Epub 2016 Oct 21.

Response to: 'Monocyte type I interferon signature in antiphospholipid syndrome is related to pro-inflammatory monocyte subsets, hydroxychloroquine and statin use' by van den Hoogen et al

Affiliations
Comment

Response to: 'Monocyte type I interferon signature in antiphospholipid syndrome is related to pro-inflammatory monocyte subsets, hydroxychloroquine and statin use' by van den Hoogen et al

Srilakshmi Yalavarthi et al. Ann Rheum Dis. 2016 Dec.
No abstract available

Keywords: Antiphospholipid Syndrome; Disease Activity; Treatment.

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Conflict of interest statement

The authors have no competing interests or conflicts to disclose.

Comment on

References

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    1. van den Hoogen LL, Fritsch-Stork RD, Versnel MA, Derksen RH, van Roon JA, Radstake TR. Monocyte type I interferon signature in antiphospholipid syndrome is related to pro-inflammatory monocyte subsets, hydroxychloroquine and statin use. Annals of the rheumatic diseases. 2016 - PubMed
    1. Perez-Sanchez C, Barbarroja N, Messineo S, Ruiz-Limon P, Rodriguez-Ariza A, Jimenez-Gomez Y, et al. Gene profiling reveals specific molecular pathways in the pathogenesis of atherosclerosis and cardiovascular disease in antiphospholipid syndrome, systemic lupus erythematosus and antiphospholipid syndrome with lupus. Annals of the rheumatic diseases. 2015 Jul;74(7):1441–1449. - PubMed
    1. Smith CK, Vivekanandan-Giri A, Tang C, Knight JS, Mathew A, Padilla RL, et al. Neutrophil extracellular trap-derived enzymes oxidize high-density lipoprotein: an additional proatherogenic mechanism in systemic lupus erythematosus. Arthritis & rheumatology. 2014 Sep;66(9):2532–2544. - PMC - PubMed
    1. Yalavarthi S, Gould TJ, Rao AN, Mazza LF, Morris AE, Nunez-Alvarez C, et al. Release of Neutrophil Extracellular Traps by Neutrophils Stimulated With Antiphospholipid Antibodies: A Newly Identified Mechanism of Thrombosis in the Antiphospholipid Syndrome. Arthritis & rheumatology. 2015 Nov;67(11):2990–3003. - PMC - PubMed

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