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Comment
. 2016 Dec 15;594(24):7157-7158.
doi: 10.1113/JP273235.

An intricate balance of muscle damage and protein synthesis: the key players in skeletal muscle hypertrophy following resistance training

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Comment

An intricate balance of muscle damage and protein synthesis: the key players in skeletal muscle hypertrophy following resistance training

Giselle Keefe et al. J Physiol. .
No abstract available

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References

    1. Brook MS, Wilkinson DJ, Mitchell WK, Lund JN, Szewczyk NJ, Greenhaff PL, Smith K & Atherton PJ (2015). Skeletal muscle hypertrophy adaptations predominate in the early stages of resistance exercise training, matching deuterium oxide‐derived measures of muscle protein synthesis and mechanistic target of rapamycin complex 1 signaling. FASEB J 29, 4485–4496. - PubMed
    1. Damas F, Phillips S, Vechin FC & Ugrinowitsch C (2015). A review of resistance training‐induced changes in skeletal muscle protein synthesis and their contribution to hypertrophy. Sports Med 45, 801–807. - PubMed
    1. Damas F, Phillips SM, Libardi CA, Vechin FC, Lixandrão ME, Jannig PR, Costa LAR, Bacurau AV, Snijders T, Parise G, Tricoli V, Roschel H & Ugrinowitsch C (2016). Resistance training‐induced changes in integrated myofibrillar protein synthesis are related to hypertrophy only after attenuation of muscle damage. J Physiol 594, 5209–5222. - PMC - PubMed
    1. Schoenfeld BJ (2010). The mechanisms of muscle hypertrophy and their application to resistance training. J Strength Cond Res 24, 2857–2872. - PubMed

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