Rebuttal from Boyett et al
- PMID: 28684595
- PMCID: PMC5625076
- DOI: 10.1152/japplphysiol.00606.2017
Rebuttal from Boyett et al
Comment on
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Exercise training-induced bradycardia: evidence for enhanced parasympathetic regulation without changes in intrinsic sinoatrial node function.J Appl Physiol (1985). 2015 Jun 1;118(11):1344-55. doi: 10.1152/japplphysiol.01111.2014. Epub 2015 Mar 6. J Appl Physiol (1985). 2015. PMID: 25749448 Free PMC article.
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Counterpoint: Exercise training-induced bradycardia: the case for enhanced parasympathetic regulation.J Appl Physiol (1985). 2017 Sep 1;123(3):686-688. doi: 10.1152/japplphysiol.00605.2017. Epub 2017 Jul 6. J Appl Physiol (1985). 2017. PMID: 28684594 No abstract available.
References
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- Billman GE, Cagnoli KL, Csepe T, Li N, Wright P, Mohler PJ, Fedorov VV. Exercise training-induced bradycardia: evidence for enhanced parasympathetic regulation without changes in intrinsic sinoatrial node function. J Appl Physiol (1985) 118: 1344–1355, 2015. doi:10.1152/japplphysiol.01111.2014. - DOI - PMC - PubMed
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- Liu W, Zi M, Jin J, Prehar S, Oceandy D, Kimura TE, Lei M, Neyses L, Weston AH, Cartwright EJ, Wang X. Cardiac-specific deletion of mkk4 reveals its role in pathological hypertrophic remodeling but not in physiological cardiac growth. Circ Res 104: 905–914, 2009. doi:10.1161/CIRCRESAHA.108.188292. - DOI - PubMed
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