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Published Erratum
. 2017 Sep;27(5):702-703.
doi: 10.1111/bpa.12512.

Erratum

No authors listed
Published Erratum

Erratum

No authors listed. Brain Pathol. 2017 Sep.
No abstract available

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Figures

Figure 2
Figure 2
In vivo effects of oleic acid (OA) supplement on brain protein levels. Western blots of mature amyloid precursor protein (APPm), immature amyloid precursor protein (APPim), soluble APPα (sAPPα), β‐site APP cleaving enzyme (BACE), β‐actin and APP–C‐terminal fragments (CTFs) (A) nicastrin (NCT), presenilin 1‐N terminal fragment (PS1‐NTF), insulin‐degrading enzyme (IDE), transthyretin (TTR), insulin‐like growth factor‐II (IGF‐II), prion protein (PrP) and β‐actin protein levels (B) in the frontal cortices of chow, low‐fat/–OA and low‐fat/+OA mice, (n = 6 for each experiment; out of six, only one animal is shown for chow, and three animals are shown for low‐fat/–OA and low‐fat/+OA mice). Respective molecular weights (kD) are shown on the left. Plots on the right show the quantitative analysis of Western blots. C. Plot shows secretase‐generated APP–C‐terminal fragment (α/β) ratios vs. OA percentage distribution. The mean +/– standard error for the mean are shown for all plots. **P < 0.01 and *P < 0.05. Abbreviation: Aβ = amyloid‐beta.
See this image and copyright information in PMC

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References

    1. Amtul Z, Westaway D, Cechetto DF, Rozmahel RF (2011) Oleic Acid Ameliorates Amyloidosis in Cellular and Mouse Models of Alzheimer's Disease. Brain Pathol 21:321–329. - PMC - PubMed

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