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Comment
. 2017 Nov;27(11):1458-1459.
doi: 10.1089/thy.2017.0281. Epub 2017 Sep 28.

Response to Krieger et al. re: "TSHR/IGF-1R Cross-Talk, Not IGF-1R Stimulating Antibodies, Mediates Graves' Ophthalmopathy Pathogenesis" (Thyroid 2017;27:746-747)

Affiliations
Comment

Response to Krieger et al. re: "TSHR/IGF-1R Cross-Talk, Not IGF-1R Stimulating Antibodies, Mediates Graves' Ophthalmopathy Pathogenesis" (Thyroid 2017;27:746-747)

Terry J Smith et al. Thyroid. 2017 Nov.
No abstract available

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References

    1. Krieger CC, Neumann S, Marcus-Samuels B, Gershengorn MC. 2017. TSHR/IGF-1R cross-talk, not IGF-1R stimulating antibodies, mediates Graves' ophthalmopathy pathogenesis. Thyroid 27:746–747 - PMC - PubMed
    1. Smith TJ, Janssen JAMJL. 2017. Building the case for insulin-like receptor-1 involvement in thyroid-associated ophthalmopathy. Front Endocrinol 7:167 - PMC - PubMed
    1. Tsui S, Naik V, Hoa N, Hwang CJ, Afifiyan NF, Sinha Hikim A, Gianoukakis AG, Douglas RS, Smith TJ. 2008. Evidence for an association between thyroid-stimulating hormone and insulin-like growth factor 1 receptors: a tale of two antigens implicated in Graves' disease. J Immunol 181:4397–4405 - PMC - PubMed
    1. Pritchard J, Han R, Horst N, Cruikshank WW, Smith TJ. 2003. Immunoglobulin activation of T cell chemoattractant expression in fibroblasts from patients with Graves' disease is mediated through the insulin-like growth factor I receptor pathway. J Immunol 170:6348–6354 - PubMed
    1. Pritchard J, Tsui S, Horst N, Cruikshank WW, Smith TJ. 2004. Synovial fibroblasts from patients with rheumatoid arthritis, like fibroblasts from Graves' disease, express high levels of IL-16 when treated with Igs against insulin-like growth factor-1 receptor. J Immunol 173:3564–3569 - PubMed

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