Commentary: Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?
- PMID: 28878736
- PMCID: PMC5572113
- DOI: 10.3389/fendo.2017.00206
Commentary: Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?
Keywords: 3-iodothyroacetic acid; 3-iodothyronamine; histamine; hypothermia; thyroid hormone derivatives; trace amines.
Comment on
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Torpor: The Rise and Fall of 3-Monoiodothyronamine from Brain to Gut-From Gut to Brain?Front Endocrinol (Lausanne). 2017 May 31;8:118. doi: 10.3389/fendo.2017.00118. eCollection 2017. Front Endocrinol (Lausanne). 2017. PMID: 28620354 Free PMC article. Review.
References
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- Manni ME, De Siena G, Saba A, Marchini M, Landucci E, Gerace E, et al. Pharmacological effects of 3-iodothyronamine (T1AM) in mice include facilitation of memory acquisition and retention and reduction of pain threshold. Br J Pharmacol (2013) 168:354–62.10.1111/j.1476-5381.2012.02137.x - DOI - PMC - PubMed
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- Laurino A, De Siena G, Saba A, Chiellini G, Landucci E, Zucchi R, et al. In the brain of mice, 3-iodothyronamine (T1AM) is converted into 3-iodothyroacetic acid (TA1) and it is included within the signaling network connecting thyroid hormone metabolites with histamine. Eur J Pharmacol (2015) 761:130–4.10.1016/j.ejphar.2015.04.038 - DOI - PubMed
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