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Editorial
. 2017 Jun;12(6):247-249.
doi: 10.2217/fvl-2017-0033. Epub 2017 Jun 8.

How do flavivirus-infected cells resist arsenite-induced stress granule formation?

Mausumi Basu  1 , Margo A Brinton  1
Affiliations
Editorial

How do flavivirus-infected cells resist arsenite-induced stress granule formation?

Mausumi Basu et al. Future Virol. 2017 Jun.
No abstract available

Keywords: flaviviruses; redox state; stress granules.

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Conflict of interest statement

Financial & competing interests disclosure M Basu is a senior scientist in the laboratory of Margo Brinton in the Department of Biology at Georgia State University. Brinton is a Regents’ Professor and received support from grants R01AI48088 and R01AI045135 from the National Institute of Allergy and Infectious Diseases, NIH. The authors have no other relevant affiliations or financial involvement with any organization or entity with a financial interest in or financial conflict with the subject matter or materials discussed in the manuscript. No writing assistance was utilized in the production of this manuscript.

References

    1. Anderson P, Kedersha N. Stress granules: the Tao of RNA triage. Trends Biochem. Sci. 2008;33(3):141–150. - PubMed
    1. Elbahesh H, Scherbik SV, Brinton MA. West Nile virus infection does not induce PKR activation in rodent cells. Virology. 2011;421(1):51–60. - PMC - PubMed
    1. Courtney SC, Scherbik SV, Stockman BM, Brinton MA. West nile virus infections suppress early viral RNA synthesis and avoid inducing the cell stress granule response. J. Virol. 2012;86(7):3647–3657. - PMC - PubMed
    1. Emara MM, Brinton MA. Interaction of TIA-1/TIAR with West Nile and dengue virus products in infected cells interferes with stress granule formation and processing body assembly. Proc. Natl Acad. Sci. USA. 2007;104(21):9041–9046. - PMC - PubMed
    1. Basu M, Courtney SC, Brinton MA. Arsenite-induced stress granule formation is inhibited by elevated levels of reduced glutathione in West Nile virus-infected cells. PLoS Pathog. 2017;13(2):e1006240. - PMC - PubMed

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