Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
. 2017 Nov;67(5):1104-1105.
doi: 10.1016/j.jhep.2017.06.001. Epub 2017 Sep 20.

Hepatic stellate cell activation and pro-fibrogenic signals

Chandrashekhar R Gandhi  1
Affiliations

Hepatic stellate cell activation and pro-fibrogenic signals

Chandrashekhar R Gandhi. J Hepatol. 2017 Nov.
No abstract available

Keywords: Activation; Fibrosis; Hepatic stellate cells; Inflammation; Liver; Signaling.

PubMed Disclaimer

Conflict of interest statement

Conflict of interest

The author declared he does not have anything to disclose regarding funding or conflict of interest with respect to this manuscript.

References

    1. Mederacke I, Hsu CC, Troeger JS, Huebener P, Mu X, Dapito DH, et al. Fate tracing reveals hepatic stellate cells as dominant contributors to liver fibrosis independent of its aetiology. Nat Commun. 2013;4:2823. - PMC - PubMed
    1. Kayama Y, Wang P, Brenner DA, Kisseleva T. In: Stellate cells in health and disease. Gandhi CR, Pinzani M, editors. Elsevier Press; 2015. pp. 87–106.
    1. Puche JE, Saiman Y, Friedman SL. Hepatic stellate cells and liver fibrosis. Compr Physiol. 2013;3:1473–1492. - PubMed
    1. Campana L, Iredale J. Matrix metalloproteinases and their inhibitors. In: Gandhi CR, Pinzani M, editors. Stellate cells in health and disease. Elsevier Press; 2015. pp. 107–124.
    1. Fabre T, Kared H, Friedman SL, Shoukry NH. IL-17A enhances the expression of profibrotic genes through upregulation of the TGF-β receptor on hepatic stellate cells in a JNK-dependent manner. J Immunol. 2014;193:3925–3933. - PMC - PubMed

Publication types