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. 2017 Nov-Dec;5(6):1808-1809.
doi: 10.1016/j.jaip.2017.08.018.

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Whitney W Stevens  1 , Lydia Suh  2 , Anju T Peters  3 , Robert P Schleimer  3
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Whitney W Stevens et al. J Allergy Clin Immunol Pract. 2017 Nov-Dec.
No abstract available

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Conflict of interest statement

Conflicts of interest: W. W. Stevens has received research support from Parker B. Francis Fellowship Program, the American Partnership for Eosinophilic Disorders/American Academy of Allergy, Asthma and Immunology HOPE Pilot Grant Award, and the National Institutes of Health’s National Center for Advancing Translational Sciences, KL2TR001424. A. T. Peters has received consultancy fees from Sanofi (advisory board membership). R. P. Schleimer has received research support from the National Institutes of Health; has received consultancy fees from Intersect ENT, GlaxoSmithKline, Allakos, Aurasense, Merck, BioMarck, Sanofi, AstraZeneca/MedImmune, Genentech, Exicure Inc., and Otsuka Inc,; and has stock in Allakos, Aurasense, BioMarck, and Exicure Inc.

Figures

FIGURE 1.
FIGURE 1.
Assessment of eosinophilia among patients with chronic rhinosinusitis with nasal polyps (CRSwNP). (A) Numbers of eosinophils in peripheral blood and (B) levels of eosinophil cationic protein (ECP) in nasal polyp tissue were measured in patients with CRSwNP (circles), CRSwNP+asthma (squares), or aspirin exacerbated respiratory disease (AERD) (diamonds). Statistical significance was determined by the Kruskal-Wallis test with post hoc analysis using Dunn’s test for multiple comparison and *P < .05 and ***P < .001. Dot pots illustrate individual data points and solid lines represent the mean ± standard error of the mean.
See this image and copyright information in PMC

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