Unconventional Signaling by Extracellular CRMP2: Possible Role as an Atypical Neurotransmitter?

Affiliations

¹ Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.
² Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA; Neuroscience Graduate Interdisciplinary Program, College of Medicine, University of Arizona, Tucson, AZ, USA; The Center for Innovation in Brain Sciences, The University of Arizona Health Sciences, Tucson, AZ, USA. Electronic address: rkhanna@email.arizona.edu.

Editorial

Aubin Moutal et al. Neuroscience. 2018.

. 2018 Apr 15:376:224-226.

doi: 10.1016/j.neuroscience.2018.02.025.

¹ Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.
² Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA; Neuroscience Graduate Interdisciplinary Program, College of Medicine, University of Arizona, Tucson, AZ, USA; The Center for Innovation in Brain Sciences, The University of Arizona Health Sciences, Tucson, AZ, USA. Electronic address: rkhanna@email.arizona.edu.

No abstract available

Keywords: CRMP2; CaV2.2; NMDA receptor; atypical neurotransmitter; neurite outgrowth; neurodegeneration; pain.

Comment on

Extracellular Application of CRMP2 Increases Cytoplasmic Calcium through NMDA Receptors.
Castillo C, Martinez JC, Longart M, García L, Hernández M, Carballo J, Rojas H, Matteo L, Casique L, Escalona JL, Rodríguez Y, Rodriguez J, Hernández D, Balbi D, Villegas R. Castillo C, et al. Neuroscience. 2018 Apr 15;376:204-223. doi: 10.1016/j.neuroscience.2018.02.002. Neuroscience. 2018. PMID: 29555037